Secondary Parkinsonism Due to a Large Anterior Cranial Fossa Meningioma

Authors

• Wissam Al-Janabi Department of Neurology, Henry Ford Health System (HFHS), Detroit, Michigan, USA and Wayne State University, School of Medicine, Detroit, Michigan, USA
• Iram Zaman Department of Neurology, Henry Ford Health System (HFHS), Detroit, Michigan, USA and Wayne State University, School of Medicine, Detroit, Michigan, USA
• Anza Memon Department of Neurology, Henry Ford Health System (HFHS), Detroit, Michigan, USA and Wayne State University, School of Medicine, Detroit, Michigan, USA

drwissamanteer@gmail.com

Keywords

Abstract

Secondary Parkinson’s disease or subacute Parkinson’s may occur after stroke, drug overdose carbon monoxide or manganese toxicity, and rarely owing to a brain tumor. Loss of dopaminergic neurons in the substansia negra pars compacta (SNc), or presence of the proteinaceous inclusions called Lewy bodies are thought to be the cause of Parkinson’s disease. Notwithstanding, in the past few decades, many case reports have been published describing Parkinson’s symptoms following either stroke, ischemia, toxicity, brain hemorrhage or rarely neoplasm.